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Lookup NU author(s): Dr Martin Picard,
Dr Amy Vincent,
Emeritus Professor Doug Turnbull
Full text for this publication is not currently held within this repository. Alternative links are provided below where available.
Clonal expansion of mtDNA deletions compromises mitochondrial function in human disease and aging, but how deleterious mtDNA genomes propagate has remained unclear. In this issue (Gitschlag et al., 2016) and in a recent Nature publication, C. elegans studies implicate the mitochondrial unfolded protein response (UPRmt) and offer mechanistic insights into this process.
Author(s): Picard M, Vincent AE, Turnbull DM
Publication type: Editorial
Publication status: Published
Journal: Cell Metabolism
Online publication date: 12/07/2016
Acceptance date: 01/01/1900
ISSN (print): 1550-4131
ISSN (electronic): 1932-7420
Publisher: Cell Press