Publications by Dr Peter Bell - ePrints

Browsing publications by Dr Peter Bell.

Newcastle Authors	Title	Year
Dr Peter Bell Dr Ella Dennis Robert Jackson Dr Anna Porter Dr Katarzyna Pirog et al.	Mesencephalic astrocyte-derived neurotrophic factor is an important factor in chondrocyte ER homeostasis	2018
Professor Michael Briggs Dr Peter Bell Dr Katarzyna Pirog	Pseudoachondroplasia and Multiple Epiphyseal Dysplasia: Molecular Genetics, Disease Mechanisms and Therapeutic Targets.	2017
Dr Peter Bell Robert Jackson Dr Katarzyna Pirog	Importance of glycosylation state and aggregation in ER-associated degradation of mutant matrilin proteins and induction of UPR	2015
Robert Jackson Dr Peter Bell Professor Michael Briggs	Molecular mechanisms provide new insight on genotype to phenotype correlations in type II collagenopathies	2015
Professor Michael Briggs Dr Peter Bell Dr Michael Wright Dr Katarzyna Pirog	New therapeutic targets in rare genetic skeletal diseases	2015
Dr Peter Bell Dr Benedetta Gualeni Dr Katarzyna Pirog	Reverse genetics approach in search for the novel MED locus	2015
Professor Michael Briggs Dr Peter Bell Dr Katarzyna Pirog	The utility of mouse models to provide information regarding the pathomolecular mechanisms in human genetic skeletal diseases: The emerging role of endoplasmic reticulum stress (Review)	2015
Dr Katarzyna Pirog Stacy Young Dr Peter Bell Professor Michael Briggs	Abnormal Chondrocyte Apoptosis in the Cartilage Growth Plate is Influenced by Genetic Background and Deletion of CHOP in a Targeted Mouse Model of Pseudoachondroplasia	2014
Professor Michael Briggs Dr Peter Bell	Genotype to phenotype correlations in cartilage oligomeric matrix protein associated chondrodysplasias	2014
Dr Benedetta Gualeni Dr Peter Bell Professor Michael Briggs	A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growth	2013
Dr Peter Bell Professor Michael Briggs	Analysis of the cartilage proteome from three different mouse models of genetic skeletal diseases reveals common and discrete disease signatures	2013
Dr Peter Bell Professor Michael Briggs	Armet/Manf and Creld2 are components of a specialized ER stress response provoked by inappropriate formation of disulphide bonds: implications for genetic skeletal diseases	2013
Dr Peter Bell Dr Katarzyna Pirog Professor Michael Briggs	Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation	2012
Dr Peter Bell Professor Michael Briggs	An unfolded protein response is the initial cellular response to the expression of mutant matrilin-3 in a mouse model of multiple epiphyseal dysplasia	2010