Publications by Dr Katarzyna Pirog - ePrints

Browsing publications by Dr Katarzyna Pirog.

Newcastle Authors	Title	Year
Dan Hayman Dr Francesca Johnson de Sousa Brito Hua Lin Yao Hao Rachel Pearson et al.	microRNA-324 mediates bone homeostasis and the regulation of osteoblast and osteoclast differentiation and activity	2025
Dr Jamie Soul Yao Hao Hua Lin Dr Katarzyna Pirog Dr Jonathan Coxhead et al.	Comparative transcriptomic analysis of articular cartilage of post-traumatic osteoarthritis mouse models.	2024
Marc Farcasanu Thais De Las Heras Ruiz Dr Katarzyna Pirog	Dynamic mechanical compression improves the chondrogenic potential of ATDC5 and primary human chondrocytes in agar hydrogels	2024
Roufaida Bouchenafa Dr Francesca Johnson de Sousa Brito Dr Katarzyna Pirog	Involvement of kinesins in skeletal dysplasia: a review	2024
Roufaida Bouchenafa Rachel Pearson Dr Katarzyna Pirog	Microtubule-Associated Motor Proteins in Skeletal Development and Health	2024
Roufaida Bouchenafa Dr Francesca Johnson de Sousa Brito Dr Katarzyna Pirog	Pro143Leu KIF22 mutation affect cartilage structure in the SEMDJL2 mice	2024
Dr Francesca Johnson de Sousa Brito Rachel Pearson Professor David Young Dr Katarzyna Pirog	Role of Asporin in chondroprotection	2024
Thais De Las Heras Ruiz Marc Farcasanu Professor David Young Dr Katarzyna Pirog	Dynamic compression dramatically improves the chondrogenic potential of ATDC5 cells in the tissue engineered model of healthy and PSACH cartilage	2021
Dan Hayman Dr Tamara Modebadze Sarah Charlton Kat Cheung Dr Jamie Soul et al.	Increased hippocampal excitability in miR-324-null mice	2021
Dr Ella Dennis Robert Jackson Dr Dimitra Tsompani Professor David Young Dr Katarzyna Pirog et al.	CRELD2 is a novel LRP1 chaperone that regulates non-canonical WNT signalling in skeletal development	2020
Sam Charlton Kat Cheung Yao Hao Dr Jamie Soul Dr Louise Reynard et al.	microRNA-seq of cartilage reveals an over-abundance of miR-140-3p which contains functional isomiRs	2020
Professor Michael Briggs Dr Ella Dennis Helen Dietmar Dr Katarzyna Pirog	New developments in chondrocyte ER-stress and related diseases	2020
Silvia Lecci Dr Beth Gibson Dr Katarzyna Pirog	Calcium activated nucleotidase 1 (CANT1) is critical for glycosaminoglycan biosynthesis in cartilage and endochondral ossification	2019
Dr Katarzyna Pirog Dr Ella Dennis Robert Jackson Dr Jamie Soul Professor Michael Briggs et al.	XBP1 signalling is essential for alleviating mutant protein aggregation in ER-stress related skeletal disease.	2019
Dr Peter Bell Dr Ella Dennis Robert Jackson Dr Anna Porter Dr Katarzyna Pirog et al.	Mesencephalic astrocyte-derived neurotrophic factor is an important factor in chondrocyte ER homeostasis	2018
Professor Michael Briggs Dr Peter Bell Dr Katarzyna Pirog	Pseudoachondroplasia and Multiple Epiphyseal Dysplasia: Molecular Genetics, Disease Mechanisms and Therapeutic Targets.	2017
Dr Ella Dennis Dr Katarzyna Pirog Professor Michael Briggs	The role of Creld2 in skeletal development and homeostasis	2016
Thais De Las Heras Ruiz Dr Katarzyna Pirog	Tissue Engineering Approaches for the Study and Therapeutic Intervention in Osteoarthritis	2016
Dr Katarzyna Pirog Professor Michael Briggs	Cartilage-Specific Ablation of XBP1 Signaling in Mouse Results in a Chondrodysplasia Characterized by Reduced Chondrocyte Proliferation and Delayed Cartilage Maturation and Mineralization	2015
Dr Peter Bell Robert Jackson Dr Katarzyna Pirog	Importance of glycosylation state and aggregation in ER-associated degradation of mutant matrilin proteins and induction of UPR	2015
Professor Michael Briggs Dr Peter Bell Dr Michael Wright Dr Katarzyna Pirog	New therapeutic targets in rare genetic skeletal diseases	2015
Dr Peter Bell Dr Benedetta Gualeni Dr Katarzyna Pirog	Reverse genetics approach in search for the novel MED locus	2015
Dr Ella Dennis Dr Katarzyna Pirog Professor Michael Briggs	The role of CRELD2 in skeletal development and homeostasis	2015
Professor Michael Briggs Dr Peter Bell Dr Katarzyna Pirog	The utility of mouse models to provide information regarding the pathomolecular mechanisms in human genetic skeletal diseases: The emerging role of endoplasmic reticulum stress (Review)	2015
Dr Beth Gibson Dr Katarzyna Pirog Professor Michael Briggs	Transgenic mice with an allelic series of aggrecan mutations model the human phenotypes	2015
Dr Katarzyna Pirog Stacy Young Dr Peter Bell Professor Michael Briggs	Abnormal Chondrocyte Apoptosis in the Cartilage Growth Plate is Influenced by Genetic Background and Deletion of CHOP in a Targeted Mouse Model of Pseudoachondroplasia	2014
Dr Katarzyna Pirog Professor Michael Briggs	Mild myopathy is associated with COMP but not MATN3 mutations in mouse models of genetic skeletal diseases	2013
Dr Katarzyna Pirog Professor Michael Briggs	Mutant chondrocytes are characterised by altered cell shape and changes to the organisation of primary cilia in a genetic mouse model of mild pseudoachondroplasia	2013
Dr Katarzyna Pirog Professor Michael Briggs	A novel form of chondrocyte stress is triggered by a COMP mutation causing pseudoachondroplasia	2012
Dr Peter Bell Dr Katarzyna Pirog Professor Michael Briggs	Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation	2012
Dr Katarzyna Pirog Professor Michael Briggs	A mouse model offers novel insights into the myopathy and tendinopathy often associated with pseudoachondroplasia and multiple epiphyseal dysplasia	2010
Dr Katarzyna Pirog Professor Michael Briggs	Molecular dissection of the unfolded protein response in hypertrophic chondrocytes	2010
Dr Katarzyna Pirog Professor Michael Briggs	Skeletal Dysplasias Associated with Mild Myopathy-A Clinical and Molecular Review	2010
Dr Katarzyna Pirog Professor Michael Briggs	Genetic background influences the severity of chondrodysplasia phenotype	2009
Dr Katarzyna Pirog Professor Michael Briggs	Genetic background influences the severity of growth plate dysplasia in a mouse model of pseudoachondroplasia	2007
Dr Katarzyna Pirog Professor Michael Briggs	Reduced cell proliferation and increased apoptosis are significant pathological mechanisms in a murine model of mild pseudoachondroplasia resulting from a mutation in the C-terminal domain of COMP	2007
Dr Katarzyna Pirog Professor Michael Briggs	T583M COMP knockin mouse - a model of mild pseudoachondroplasia resulting from C-terminal COMP mutation	2006
Dr Katarzyna Pirog	Changes in Bcl-2 expression in vaccinia virus-infected human peripheral blood monocytes	2005
Dr Katarzyna Pirog	Terapia genowa (Gene therapy)	2002