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Browsing publications by Dr Katarzyna Pirog.

Newcastle AuthorsTitleYearFull text
Dan Hayman
Dr Tamara Modebadze
Sarah Charlton
Kat Cheung
Dr Jamie Soul
et al.
Increased hippocampal excitability in miR-324-null mice2021
Dr Ella Dennis
Robert Jackson
Dr Dimitra Tsompani
Professor David Young
Dr Katarzyna Pirog
et al.
CRELD2 is a novel LRP1 chaperone that regulates non-canonical WNT signalling in skeletal development2020
Sam Charlton
Kat Cheung
Yao Hao
Dr Jamie Soul
Dr Louise Reynard
et al.
microRNA-seq of cartilage reveals an over-abundance of miR-140-3p which contains functional isomiRs2020
Professor Michael Briggs
Dr Ella Dennis
Helen Dietmar
Dr Katarzyna Pirog
New developments in chondrocyte ER-stress and related diseases2020
Silvia Lecci
Dr Beth Gibson
Dr Katarzyna Pirog
Calcium activated nucleotidase 1 (CANT1) is critical for glycosaminoglycan biosynthesis in cartilage and endochondral ossification2019
Dr Katarzyna Pirog
Dr Ella Dennis
Robert Jackson
Dr Jamie Soul
Professor Michael Briggs
et al.
XBP1 signalling is essential for alleviating mutant protein aggregation in ER-stress related skeletal disease.2019
Dr Peter Bell
Dr Ella Dennis
Robert Jackson
Dr Anna Porter
Dr Katarzyna Pirog
et al.
Mesencephalic astrocyte-derived neurotrophic factor is an important factor in chondrocyte ER homeostasis2018
Professor Michael Briggs
Dr Peter Bell
Dr Katarzyna Pirog
Pseudoachondroplasia and Multiple Epiphyseal Dysplasia: Molecular Genetics, Disease Mechanisms and Therapeutic Targets.2017
Dr Ella Dennis
Dr Katarzyna Pirog
Professor Michael Briggs
The role of Creld2 in skeletal development and homeostasis2016
Thais De Las Heras Ruiz
Dr Katarzyna Pirog
Tissue Engineering Approaches for the Study and Therapeutic Intervention in Osteoarthritis2016
Dr Katarzyna Pirog
Professor Michael Briggs
Cartilage-Specific Ablation of XBP1 Signaling in Mouse Results in a Chondrodysplasia Characterized by Reduced Chondrocyte Proliferation and Delayed Cartilage Maturation and Mineralization2015
Dr Peter Bell
Robert Jackson
Dr Katarzyna Pirog
Importance of glycosylation state and aggregation in ER-associated degradation of mutant matrilin proteins and induction of UPR2015
Professor Michael Briggs
Dr Peter Bell
Dr Michael Wright
Dr Katarzyna Pirog
New therapeutic targets in rare genetic skeletal diseases2015
Dr Peter Bell
Dr Benedetta Gualeni
Dr Katarzyna Pirog
Reverse genetics approach in search for the novel MED locus2015
Dr Ella Dennis
Dr Katarzyna Pirog
Professor Michael Briggs
The role of CRELD2 in skeletal development and homeostasis2015
Professor Michael Briggs
Dr Peter Bell
Dr Katarzyna Pirog
The utility of mouse models to provide information regarding the pathomolecular mechanisms in human genetic skeletal diseases: The emerging role of endoplasmic reticulum stress (Review)2015
Dr Beth Gibson
Dr Katarzyna Pirog
Professor Michael Briggs
Transgenic mice with an allelic series of aggrecan mutations model the human phenotypes2015
Dr Katarzyna Pirog
Stacy Young
Dr Peter Bell
Professor Michael Briggs
Abnormal Chondrocyte Apoptosis in the Cartilage Growth Plate is Influenced by Genetic Background and Deletion of CHOP in a Targeted Mouse Model of Pseudoachondroplasia2014
Dr Katarzyna Pirog
Professor Michael Briggs
Mild myopathy is associated with COMP but not MATN3 mutations in mouse models of genetic skeletal diseases2013
Dr Katarzyna Pirog
Professor Michael Briggs
Mutant chondrocytes are characterised by altered cell shape and changes to the organisation of primary cilia in a genetic mouse model of mild pseudoachondroplasia2013
Dr Katarzyna Pirog
Professor Michael Briggs
A novel form of chondrocyte stress is triggered by a COMP mutation causing pseudoachondroplasia2012
Dr Peter Bell
Dr Katarzyna Pirog
Professor Michael Briggs
Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation2012
Dr Katarzyna Pirog
Professor Michael Briggs
A mouse model offers novel insights into the myopathy and tendinopathy often associated with pseudoachondroplasia and multiple epiphyseal dysplasia2010
Dr Katarzyna Pirog
Professor Michael Briggs
Skeletal Dysplasias Associated with Mild Myopathy-A Clinical and Molecular Review2010
Dr Katarzyna Pirog
Professor Michael Briggs
Reduced cell proliferation and increased apoptosis are significant pathological mechanisms in a murine model of mild pseudoachondroplasia resulting from a mutation in the C-terminal domain of COMP2007
Dr Katarzyna Pirog
Changes in Bcl-2 expression in vaccinia virus-infected human peripheral blood monocytes2005
Dr Katarzyna Pirog
Terapia genowa (Gene therapy)2002